People

Eric C. Beyer, MD PhD

  • Professor of Pediatrics
  • Clinical Interests: Anemias, Childhood Blood Cancers, Childhood Cancers, Childhood Leukemia, Pediatric Hematology, Sickle Cell Disease
  • Research and Scholarly Interests: Arrhythmia, Cardiac, Cancer, Cataract, Cell Biology, Connexins, Endothelial Cells, Exosomes, Extracellular vessicles, Gap Junctions, Intercellular Junctions, Ion Channel, Lens, Eye, Sickle Cell Disease, Translational Medical Research
  • Websites: My Google Scholar site, Research Network Profile
  • Contact: ecbeyer@uchicago.edu
  • Graduate Program: Committee on Cancer Biology

Professor of Pediatrics

Section of Hematology/Oncology and Stem Cell Transplantation

University of Chicago
AB - Biological Sciences

UCSD
PHD - Physiology and Pharmacology

UCSD
MD - Medicine

Children's Hospital
Boston
Internship and Residency - Pediatrics

Children's Hospital and Dana Farber Cancer Center
Boston
Fellowship - Pediatric Hematology/Oncology

Impaired cellular trafficking of a thrombomodulin mutant causes severe bleeding, thrombosis, and atypical hemolytic uremic syndrome.
Impaired cellular trafficking of a thrombomodulin mutant causes severe bleeding, thrombosis, and atypical hemolytic uremic syndrome. Blood Vessel Thromb Hemost. 2026 Aug; 3(3):100182.
PMID: 42569427

Hypoxia Increases Connexin46 and Connexin43 Levels in KNS-42 Glioblastoma Cells.
Hypoxia Increases Connexin46 and Connexin43 Levels in KNS-42 Glioblastoma Cells. Int J Mol Sci. 2026 Mar 21; 27(6).
PMID: 41898710

Plasma Levels of MicroRNA Let-7c-5p May Predict Risk of Acute Chest Syndrome in Patients with Sickle Cell Disease.
Plasma Levels of MicroRNA Let-7c-5p May Predict Risk of Acute Chest Syndrome in Patients with Sickle Cell Disease. Int J Mol Sci. 2025 Apr 18; 26(8).
PMID: 40332489

Connexin channels and hemichannels are modulated differently by charge reversal at residues forming the intracellular pocket.
Connexin channels and hemichannels are modulated differently by charge reversal at residues forming the intracellular pocket. Biol Res. 2024 May 23; 57(1):31.
PMID: 38783330

Pediatric cataracts of different etiologies contain insoluble, calcified particles.
Pediatric cataracts of different etiologies contain insoluble, calcified particles. Front Ophthalmol (Lausanne). 2023; 3.
PMID: 37441058

A crystallin mutant cataract with mineral deposits.
A crystallin mutant cataract with mineral deposits. J Biol Chem. 2023 08; 299(8):104935.
PMID: 37331601

Editorial: Ion channels, pumps, and transporters in lens physiology and disease.
Editorial: Ion channels, pumps, and transporters in lens physiology and disease. Front Physiol. 2022; 13:1071215.
PMID: 36406990

Loss of fiber cell communication may contribute to the development of cataracts of many different etiologies.
Loss of fiber cell communication may contribute to the development of cataracts of many different etiologies. Front Physiol. 2022; 13:989524.
PMID: 36171977

Levels and Modifications of Both Lens Fiber Cell Connexins Are Affected in Connexin Mutant Mice.
Levels and Modifications of Both Lens Fiber Cell Connexins Are Affected in Connexin Mutant Mice. Cells. 2022 09 07; 11(18).
PMID: 36139360

Connexin Mutants Cause Cataracts Through Deposition of Apatite.
Connexin Mutants Cause Cataracts Through Deposition of Apatite. Front Cell Dev Biol. 2022; 10:951231.
PMID: 35938173

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American Society for Clinical Investigation

Society for Pediatric Research

Fellow, American Heart Association

Fellow, Association for Research in Vision and Ophthalmology

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